Noelle Buonaccorsi, MD
PGY-4 Resident
Medical School: Medical College of Wisconsin
Year of Graduation: 2009
Email: jbuonacc@mcw.edu
I am originally from Springfield, Virginia (a suburb of Washington D.C.) and moved to Wisconsin for medical school. I love living here because the housing prices are much more reasonable than on the east coast, there is much less traffic, and the summers are beautiful. My husband and I have three cats (Sweetie Pie, Bastet, and Spot) and we live in Waukesha, a suburb of Milwaukee. My experience as a resident in pathology at MCW has been wonderful, the attendings are very approachable and it is a great learning environment.
I graduated in 2000 with a bachelor’s degree in Psychology from George Mason University. I worked in the healthcare field for several years and then went back to school to take the prerequisites needed for medical school. At the Medical College of Wisconsin I participated in the Medical Student Summer Research Program designed for students interested in pursuing research during the summer between the first and second year of medical school. I worked in the Cardiovascular Research Center studying the effects of arachidonic acid metabolites on apoptosis in mouse cardiomyocytes and bovine pulmonary artery endothelial cells. I continued my research throughout medical school and was awarded the Honors in Research distinction on my diploma (2009) for the acceptance of a thesis detailing my research.
Publications and Presentations
Anuradha Dhanasekaran, Sreedhar Bodiga, Stephanie Gruenloh, Ying Gao, Laurel Dunn, John R Falck, J Noelle Buonaccorsi, Meetha Medhora, Elizabeth R Jacobs. 20-HETE increases survival and decreases apoptosis in pulmonary arteries and pulmonary artery endothelial cells. American journal of physiology. Heart and circulatory physiology. 2009 Mar
Anuradha Dhanasekaran, Stephanie K Gruenloh, J Noelle Buonaccorsi, Rong Zhang, Garrett j Gross, John R Falck, Paresh K Patel, Elizabeth R Jacobs, Meetha Medhora. Multiple antiapoptotic targets of the PI3K/Akt survival pathway are activated by epoxyeicosatrienoic acids to protect cardiomyocytes from hypoxia/anoxia. American journal of physiology. Heart and circulatory physiology. 2008 Feb