MCW Cardiovascular Center Faculty Labs
Ivor J. Benjamin MD, FAHA, FACC, is a Professor of Medicine at Froedtert Hospital, and the Medical College of Wisconsin. A board-certified specialist and consultant in internal medicine and cardiology, Dr. Benjamin’s clinical interests are general cardiology, inheritable heart failure, and myocardial infarction.
Dr. Auchampach is a professor in the Department of Pharmacology. He leads an NIH-funded laboratory focused on ischemic heart disease, cardiac regeneration, and drug development.
The Beyer lab studies the complex relationship and physiological effects of vascular stress response with and aging.
Dr. Freed’s research primarily focuses on the role that sphingolipids have in the development of endothelial dysfunction in the human microcirculation. Elevated plasma levels of ceramide, a prototypical sphingolipid, is now considered an independent risk factor for major adverse cardiovascular events in otherwise healthy people.
Dr. Geurts is an Associate Professor in the Department of Physiology, Cardiovascular Research Center and Human and Molecular Genetics Center. He leads an NIH-funded research team focused on innovating novel approaches to genetic engineering and creating animal and cell models of complex genetic human diseases.
The research in Dr. Goldspink's laboratory is focused on understanding the actions of IGF-1 isoforms in the heart and other tissues.
Dr. Gutterman is the Northwestern Mutual Professor of Cardiology. He is actively involved in clinical practice, supervises a NIH funded research laboratory and provides senior administrative oversight of research administration at the Medical College of Wisconsin.
The major focus of our laboratory is to understand mechanisms regulating the cerebral circulation under physiologic and pathophysiologic conditions.
The primary goal of the Regner laboratory is to investigate the cellular, molecular, and genetic mechanisms involved in ischemic acute kidney injury (AKI) and kidney repair.
Dr. Sorci-Thomas's basic research laboratory focuses on the molecular mechanisms involved in high density lipoprotein apoA-I-mediated protection against the progression of human disease. The lab is particularly interested in the role apoA-I plays in modulating cellular cholesterol levels and the impact of this process on immune cell function and the development of atherosclerosis, autoimmunity and obesity.
We study heart disease at varying levels of biological complexity including patients, animal models and individual heart cells to obtain insights into disease mechanisms. We are currently investigating the cellular and molecular mechanisms that cause cardiomyopathy in patients with muscular dystrophy.
Dr. Michael E. Widlansky's human vascular research laboratory has been formed to foster collaboration with investigators from other disciplines interested in the impact of vascular function on disease states relevant to their fields of interest.
The overall emphasis of Dr. David X. Zhang's research is to understand the cellular mechanisms by which the endothelium regulates blood vessel tone in both normal physiological conditions and disease states, such as ischemic heart disease and hypertension.