Oleg Palygin, PhD
Assistant Professor
Locations
- Physiology
Contact Information
Education
PhD, Bogomoletz Institute of Physiology, 2004
MS, Taras Shevchenko National University, 1999
Postdoctoral Scholar, Molecular Physiology and Biophysics, University of Iowa
Postdoctoral Fellow, School of Life Sciences, Warwick University
MS, Taras Shevchenko National University, 1999
Postdoctoral Scholar, Molecular Physiology and Biophysics, University of Iowa
Postdoctoral Fellow, School of Life Sciences, Warwick University
Research Interests
Renal Physiology | Neurophysiology | Cardiovascular Physiology | Genetics and Genomics
The capacity of the human kidney to control and regulate cardiovascular health is enormous and has been implicated in many of the physiological factors associated with the general health status. Today, a greater understanding has been achieved of the cellular and molecular mechanisms underlying the processes of renal function, and how these processes may become disordered in chronic kidney disease states such as hypertension, polycystic kidney and diabetes mellitus.
Our laboratory is interested in several aspects of the kidney: its ability to control and regulate body electrolytes, its vulnerability to genetically related mutations, oxidative stress, albuminuria and the role that transport potassium channels, protease activated receptors and the purines like ATP play in these processes.
The capacity of the human kidney to control and regulate cardiovascular health is enormous and has been implicated in many of the physiological factors associated with the general health status. Today, a greater understanding has been achieved of the cellular and molecular mechanisms underlying the processes of renal function, and how these processes may become disordered in chronic kidney disease states such as hypertension, polycystic kidney and diabetes mellitus.
Our laboratory is interested in several aspects of the kidney: its ability to control and regulate body electrolytes, its vulnerability to genetically related mutations, oxidative stress, albuminuria and the role that transport potassium channels, protease activated receptors and the purines like ATP play in these processes.